Ana sayfa | MS’in Derinliği  | MS Slide Resource

Foreword

Complex traits are disorders determined by interactions among genes, environment and chance. They include conditions such as atherosclerosis, hypertension and cancer - the most common causes of mortality and morbidity in the developed world. Most chronic autoimmune diseases appear to be complex traits also. Multiple sclerosis, a disease which most investigators consider to be autoimmune in nature, epitomizes the complexity of gene/environment/chance interactions. In the text that follows, we summarize and interpret the plentiful epidemiological evidence gathered on multiple sclerosis with an attempt to place these into the general context of aetiology.

  George C. Ebers

Guest editor

Dr George C. Ebers graduated in medicine from the University of Toronto in 1970. He interned at McGill and spent a year as a family doctor in Strathroy, Ontario. He trained in Neurology at Cornell in 1972 to 1975, and from 1975 to 1977 was a post-doctoral fellow at Rockerfeller University. He was a visiting scholar at Green College, Oxford in 1986-87. He has worked for many years in the areas of epidemiology, clinical trials and molecular genetics, with a particular focus on multiple sclerosis. His laboratory has made important contributions to the understanding of this disease as well as a number of neuromuscular diseases. He serves on numerous editorial boards and is the author and co-author of some 150 scientific papers.

Produced as an educational service from Schering AG.

First Edition 1995.

The opinions expressed in this educational programme are not necessarily those of Schering AG.

©1995 Schering AG. All rights reserved, including that of translation into other languages. No part of this book may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or any information storage and retrieval systems, without permission from Schering AG.

Bu sayfadaki bilginin en son güncellendiği/doğrulandığı tarih:

11/09/2001

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